Capillary leak due to differentiation syndrome following the treatment of acute promyelocytic leukemia with retinoic acid or arsenic trioxide occurs in ∼25% of cases
Edema / chemically induced
The term capillary leak syndrome (CLS) describes the manifestations associated with an increased capillary permeability to proteins leading to an escape of
Capillary leak syndrome (CLS) or vascular leak syndrome (VLS) was first described by Clarkson in 1960 [1]
Systemic capillary leak syndrome (SCLS), initially described by Clarkson, is a rare disease characterized by reversible plasma extravasation, circulatory collapse
Here, we report a case of ISCLS with Systemic capillary leak syndrome (SCLS) is a rare disease characterized by shock caused by capillary hyperpermeability
In our case, there was generalized edema, increased The leak phase follows with oliguria, arterial hypotension and rapid development of edema of the face or the upper limbs but sparing the lungs
This entity's hallmark is a double clinico-biological paradox (during leak phase) associating diffuse pitting edema with
Bayard Clarkson described a woman experiencing sporadic, recurrent episodes of shock and anasarca
Figure 2| Management of capillary leak syndrome
Sepsis capillary leak syndrome (SCLS) is an independent prognostic factor for poor sepsis outcome
2011 4
sepsis (common) autoimmune diseases; differentiation syndrome; engraftment syndrome;
1 Depletion of the intravascular volume leads to manifestations of hypovolemia, including hypotension, shock, and acute kidney injury (AKI)
We describe the rare association between secondary capillary leak syndrome (SCLS) and autoimmune Management of Engraftment Syndrome and Capillary Leak Syndrome
Introduction
Capillary leak syndrome is a rare and potentially life-threatening condition caused by a shift of intravascular fluid and proteins to the interstitial space
Systemic capillary leak syndrome (SCLS) is a rare disorder characterized by severe hypotension, hemoconcentration, and hypoalbuminemia due to extravasation of plasma and proteins into the interstitial space as a result of endothelial dysfunction